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发布于:2018-6-14 19:25:11  访问:3 次 回复:0 篇
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Do You Have A Smoothened Agonist Quandary ? Well Read This One
Continuous variables between subjects with and without HP infection were compared using student‘s t-tests and categorical variables using chi-square tests. Multiple logistic regression models were used to analyze the relationship between obesity and HP infection with adjustment for participants‘ characteristics. Subjects with HP infection had a higher prevalence of obesity, current smoking and current alcohol use, but a lower value of eGFR as compared to subjects without it (Table?1). Based on the multiple logistic regression, obesity (OR: 1.89, 95% CI: 1.04�C3.45, P?=?.037) and current smoking (OR: 3.26, 95% CI: 1.15�C9.24, P?=?.026) were positively associated with HP infection, while age ��65 vs <65?years (OR: 0.57, 95% CI: 0.35�C0.94, Smoothened Agonist P?=?.027) and eGFR (OR: 0.99, 95% CI: 0.97�C1.00, P?=?.046) were inversely related to HP infection after adjusting for participants‘ characteristics. To the best of our knowledge, this is the first study focusing on the relationship between obesity and HP infection in elderly subjects. Obesity was positively associated with an increased risk of HP infection SP 600125 in the elderly subjects examined in the current work. The subgroup analysis carried out in Kopacova et?al. study[3] showed the trend of a positive but nonsignificant association between HP infection and obesity in the elderly. This discrepancy may be related to some differences in selection of subjects, definition of obesity, and diagnostic methods of HP infection. The mechanisms for the association between obesity and HP infection may involve the deterioration of immunity. The bactericidal capacity of granulocyte, maturation of monocytes into macrophages and cytotoxic function of natural killer cells have all been shown to be reduced in morbidly obese subjects.[5] Besides, HP-infected subjects have lower serum leptin levels,[6] which stimulate hunger,[7] and overfeeding may be involved in the mechanisms of central leptin resistance seen in obesity.[8] However, the relationship among leptin, obesity, and HP infection needs further investigation. The first limitation of this study is that biopsy was not performed in every subject, resulting in the potential risk of a selection bias. Second, there learn more are a variety of methods for diagnosis of HP infection, but there is no single test that can be considered the gold standard.[9] However, with properly trained personnel, histopathology has greater sensitivity and specificity than serological tests,[9] and the latter may remain positive results even 4?years after successful treatment,[10] and thus may not represent current infection status. Conflict of Interest: The editor in chief has reviewed the conflict of interest checklist provided by the authors and has determined that the authors have no financial or any other kind of personal conflicts with this paper. This work was supported by grant NCKUHFM-101-002 from the Department of Family Medicine, National Cheng-Kung University Hospital.
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